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雞流感H7N9病毒快檢卡(膠體金法):日本富士(瑞必歐)、日本生研、美國(guó)BD、美國(guó)NovaBios、美國(guó)binaxNOW、英國(guó)clearview、凱必利、廣州創(chuàng)侖等。歡迎大家,廣州健侖生物科技有限公司

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雞流感H7N9病毒快檢卡(膠體金法)

廣州健侖生物科技有限公司

廣州健侖長(zhǎng)期供應(yīng)各種PCR試劑盒,主要代理進(jìn)口和國(guó)產(chǎn)品牌的流行病毒PCR檢測(cè)試劑盒。例如:甲乙型流感病毒核酸檢測(cè)試劑盒、黃熱病毒核酸檢測(cè)試劑盒、諾如病毒核酸檢測(cè)試劑盒、登革病毒核酸檢測(cè)試劑盒、基孔肯雅病毒核酸檢測(cè)試劑盒、結(jié)核桿菌核酸病毒檢測(cè)試劑盒、孢疹病毒核算檢測(cè)試劑盒、西尼羅河病毒PCR檢測(cè)試劑盒、呼吸道合胞病毒核酸檢測(cè)試劑盒、冠狀病毒PCR檢測(cè)試劑盒等等。蟲(chóng)媒體染病系列、呼吸道病原體系列、發(fā)熱伴出疹系列、消化道及食源感染系列。

廣州健侖長(zhǎng)期供應(yīng)各種流感檢測(cè)試劑,包括進(jìn)口和國(guó)產(chǎn)的品牌,主要包括日本富士瑞必歐、日本生研、美國(guó)BD、美國(guó)NovaBios、美國(guó)binaxNOW、英國(guó)clearview、凱必利、廣州創(chuàng)侖等主流品牌。

主要檢測(cè):甲型流感病毒檢測(cè)試劑、乙型流感病毒檢測(cè)試劑、甲乙型流感病毒檢測(cè)試劑、A+B流感病毒檢測(cè)試劑盒、流感病毒抗原快速檢測(cè)卡、流感病毒抗體快速檢測(cè)試劑盒、流感快速檢測(cè)試劑 c1c2。

本病潛伏期的長(zhǎng)短隨家禽所感染病毒的毒力、病毒數(shù)量、感染途徑等不同有較大差異,由幾小時(shí)至幾天不等。其病型根據(jù)雞得禽流感的癥狀及病理變化不同可分為急性敗血型、急性呼吸道型和非典型三類(lèi)。  急性敗血型禽流感(典型禽流感)  病禽主要的癥狀表現(xiàn)為高度沉郁、昏睡,張口喘氣,流淚流涕(在水禽有時(shí)可見(jiàn)眼鼻流出膿樣液體),冠髯發(fā)紺、出血,頭頸部腫大,急性死亡。部分病例出現(xiàn)共濟(jì)失調(diào)、震顫、偏頭、扭頸等神經(jīng)癥狀。病理變化特征是眼角膜混濁,眼結(jié)膜出血、潰瘍;翅膀、嗉囊部皮膚表面有紅黑色斑塊狀出血等;還常見(jiàn)腳脛出血(腳脛鱗片出現(xiàn)紅褐色出血斑塊)、水腫;皮下水腫(尤其是頭頸、皮下)或呈凍膠樣浸潤(rùn);肺臟出血水腫,脾臟有灰白色斑點(diǎn)樣壞死,胰臟有褐色斑點(diǎn)樣出血、變性、壞死;法氏囊出血;從口腔至泄殖腔整個(gè)消化道黏膜出血、潰瘍或有灰白色斑點(diǎn)、條紋樣膜狀物(壞死性偽膜),其他組織器官亦可能有出血,并??梢?jiàn)有明顯的纖維素性腹膜炎、氣囊炎等。有的病雞心肌有灰白色壞死性條紋。  急性呼吸道型禽流感(典型禽流感)  病禽主要表現(xiàn)為流淚流涕、呼吸急促、咳嗽、打噴嚏,鼻竇腫脹,下痢,部分發(fā)生死亡(在與新城疫病毒或其他禽I型副黏病毒株合并感染時(shí),死亡率較高)。主要病理變化為喉頭氣管出血,鼻竇積聚分泌物,眼結(jié)膜水腫出血,有時(shí)亦見(jiàn)類(lèi)似急性敗血型禽流感的病理變化。  非典型禽流感  病禽一般表現(xiàn)為流淚、咳嗽、喘氣、下痢,產(chǎn)蛋率大幅度下降(下降幅度為50%~80%),并發(fā)生零星死亡。大體病理變化為鼻竇、氣管、氣囊、腸道有一些滲出性炎癥,有時(shí)見(jiàn)氣囊有纖維素性滲出,囊壁增厚,母禽發(fā)生卵黃性腹膜炎。

雞流感H7N9病毒快檢卡(膠體金法)

The length of the incubation period of the disease with the virulence of the virus infected poultry, the number of viruses, different routes of infection are quite different, ranging from a few hours to several days. According to the chicken infected with the symptoms and pathological changes of the bird flu can be divided into acute sepsis, acute respiratory and atypical three categories. Acute septic (bird flu) The main symptoms of sick birds are highly depressed, lethargic, breathless, runny nose (pus-like fluid sometimes seen in waterfowls), cyanosis of the crown, bleeding, head and neck Swollen, acute death. Some cases of ataxia, tremor, migraine, torsion neck and other neurological symptoms. Pathological changes are characterized by corneal opacity, conjunctival hemorrhage, ulceration; red and black plaque-like hemorrhages on the skin of the wings and cuffs; and common hemorrhoid bleeding (edema) Edema (especially the head and neck, chest subcutaneous) or was like jelly-like infiltration; pulmonary hemorrhage edema, spleen gray spot-like necrosis, pancreatic brown spot-like hemorrhage, degeneration and necrosis; Bursal disease hemorrhage; from oral to cloacal digestion Road mucosal bleeding, ulcers or gray spots, stripe-like membranous (necrotic pseudomembranous), other tissues and organs may also have bleeding, and often have obvious cellulite peritonitis, air sac inflammation and so on. Some sick chicken gray necrotic stripes. Acute respiratory tract avian influenza (typical bird flu) is mainly manifested as tears, runny nose, shortness of breath, cough, sneezing, swelling of the sinuses, diarrhea, and death in some cases (in the case of Newcastle disease virus or other avian Paramyxovirus Type I strains Combined infection, the mortality rate higher). The main pathological changes were laryngeal tracheal hemorrhage, secretions of the sinuses, conjunctival edema and sometimes pathological changes similar to those of acute septic cases of bird flu. Typical cases of atypical avian influenza birds show tearing, coughing, gasping and diarrhea, and the egg production rate has dropped drastically (by 50% -80%), resulting in sporadic deaths. The general pathological changes for the sinus, trachea, balloon, intestinal exudative inflammation, and sometimes see the air bag with cellulose exudate, wall thickening, egg yolk peritonitis occurs in the fallopian tubes, tubal inflammation exudate.

 

 

我司還提供其它進(jìn)口或國(guó)產(chǎn)試劑盒:登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲(chóng)病、違禁品濫用、肺炎球菌、軍團(tuán)菌、化妝品檢測(cè)、食品安全檢測(cè)等試劑盒以及日本生研細(xì)菌分型診斷血清、德國(guó)SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。

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雞流感H7N9病毒快檢卡(膠體金法)

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【公司名稱(chēng)】 廣州健侖生物科技有限公司
【市場(chǎng)部】     歐

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【騰訊  】 
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-103室

 

1)單純擴(kuò)散:脂溶性物質(zhì)由膜的高濃度側(cè)向低濃度側(cè)的擴(kuò)散過(guò)程,稱(chēng)為單純擴(kuò)散。不耗能,不需要載體。如:水、尿素、二氧化碳等.
2)協(xié)助擴(kuò)散:非脂溶性物質(zhì)在膜蛋白的幫助下,順濃度差或電位差跨膜擴(kuò)散的過(guò)程,稱(chēng)為協(xié)助擴(kuò)散。不耗能,但是需要載體。協(xié)助擴(kuò)散的三個(gè)特點(diǎn):1、特異性:記憶中離子通道或載體一般只轉(zhuǎn)運(yùn)一種物質(zhì)。2、飽和性:即當(dāng)被轉(zhuǎn)運(yùn)物質(zhì)增加到一定限度時(shí),轉(zhuǎn)運(yùn)速率不再隨之增加,這是由于離子通道或載體的數(shù)量有限的緣故。3、競(jìng)爭(zhēng)性抑制:記憶中離子通道或載體同時(shí)轉(zhuǎn)運(yùn)兩種或兩種以上物質(zhì)時(shí),一種物質(zhì)濃度增加,將削弱對(duì)另一種物質(zhì)的轉(zhuǎn)運(yùn)。4.膜蛋白的分類(lèi):1 通道蛋白 2 門(mén)通道蛋白 3特化蛋白(通過(guò)接觸改變自身構(gòu)象來(lái)進(jìn)行轉(zhuǎn)運(yùn))如:葡萄糖進(jìn)入紅細(xì)胞。
自由擴(kuò)散和協(xié)助擴(kuò)散都是順濃度差進(jìn)行的,細(xì)胞本身不消耗能量,均屬于被動(dòng)轉(zhuǎn)運(yùn)(被動(dòng)運(yùn)輸)。
3)主動(dòng)運(yùn)輸:離子或小分子物質(zhì)在膜上“泵”的作用下,被逆濃度差或逆電位差的跨膜轉(zhuǎn)運(yùn)過(guò)程,稱(chēng)為主動(dòng)轉(zhuǎn)運(yùn)(主動(dòng)運(yùn)輸)。主動(dòng)運(yùn)輸需要消耗大量熱量并且需要載體。有選擇透過(guò)性。如:碘進(jìn)入海帶、葡萄糖進(jìn)入除紅細(xì)胞以外的細(xì)胞。
4)胞吞胞吐:是轉(zhuǎn)運(yùn)大分子或團(tuán)塊物質(zhì)的有效方式。物質(zhì)通過(guò)細(xì)胞膜的運(yùn)動(dòng)從細(xì)胞外進(jìn)入細(xì)胞內(nèi)的過(guò)程,稱(chēng)胞吞。包括吞噬和吞飲。液態(tài)物質(zhì)入胞為吞飲,如小腸上皮對(duì)營(yíng)養(yǎng)物質(zhì)的吸收;固體物質(zhì)入胞為吞噬,如粒細(xì)胞吞噬細(xì)菌的過(guò)程。胞吐是通過(guò)細(xì)胞膜的運(yùn)動(dòng)從細(xì)胞內(nèi)派到細(xì)胞外的過(guò)程。細(xì)胞的代謝產(chǎn)物及腺細(xì)胞的分泌物都是以胞吐作用完成的,需要消耗能量。
5)細(xì)胞膜的受體功能:受體是細(xì)胞識(shí)別和結(jié)核化學(xué)信息的特殊結(jié)構(gòu),其本質(zhì)是蛋白質(zhì)(糖蛋白)。
補(bǔ)充:
1)細(xì)胞是物質(zhì)從無(wú)生命到有生命的zui小單元(且不論病毒),深度分析細(xì)胞的能量流動(dòng)有助于了解 生命物質(zhì)與非生命物質(zhì)的 根本區(qū)別。
2)主動(dòng)運(yùn)輸和被動(dòng)運(yùn)輸屬于穿膜運(yùn)輸,直接穿膜,*于小分子與離子。而入胞與出胞作用(內(nèi)吞與外排)屬于膜泡運(yùn)輸,不穿膜,限于大分子。1) Simple diffusion: The fat-soluble substance is diffused from the high-concentration side of the membrane to the low-concentration side, which is called simple diffusion. No energy, no carrier needed. Such as: water, urea, carbon dioxide and so on.
2) Assisting in diffusion: The process by which non-fat-soluble substances diffuse across the membrane in poor cis concentration or potential difference, with the help of membrane proteins, to assist diffusion. Not energy, but need a carrier. Three characteristics to help diffusion: 1, specificity: ion channels or memory in memory generally only transport a substance. Saturation: when the transported substance increases to a certain limit, the transport rate no longer increases, which is due to the limited number of ion channels or carriers. 3, competitive inhibition: Memory channels or carriers ion transport two or more substances simultaneously, an increase in the concentration of a substance, will weaken the transport of another substance. 4. Classification of membrane proteins: 1-channel protein 2-channel protein 3 specialized proteins (transporters that change their conformation by contact) eg glucose into erythrocytes.
Both free proliferation and assisting diffusion are carried out with the difference in cis concentration, and the cells themselves do not consume energy and both belong to passive transport (passive transport).
3) active transport: ions or small molecules in the membrane under the "pump" under the inverse density difference or reverse translocation of poor transport process, known as active transport (active transport). Active transport takes a lot of heat and requires a carrier. There is a choice of sex. Such as: iodine into kelp, glucose into cells other than red blood cells.
4) Endocytosis: It is an effective way to transport macromolecules or lumps. Matter through the cell membrane movement from the cell into the cell process, called endocytosis. Including phagocytosis and swallowing. Into the liquid material for swallowing drink, such as the intestinal epithelium of nutrients absorption; solid matter into the cells for phagocytosis, such as granulocytes phagocytosis of bacteria. Exocytosis is the process of sending cells extracellularly through the movement of cell membranes. Cell metabolites and glandular cell secretions are exocytosis to complete the role, the need to consume energy.
5) Receptor function of cell membrane: The receptor is a special structure of chemical information of cell recognition and tuberculosis, and its essence is protein (glycoprotein).
supplement:
1) Cells are the smallest unit of matter from inanimate to living (and regardless of virus). In-depth analysis of cellular energy flow helps to understand the fundamental difference between living and non-living matter.
2) active transport and passive transport are transmembrane transport, direct through the membrane, limited to small molecules and ions. The entry and exocytosis (endocytosis and efflux) belong to the vesicle transport, do not wear the membrane, limited to macromolecules.

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